Health issues
Instead of writing the history of this breed which each breeder knows and every new comer can find on the many other homepages, I decided to write something which benefits the breed.
80tiesAs a breeder, in the late and mid 80ties, El Ubaid’s was being created. The first purchased salukis had terrible temperaments very hysteric shy, cancer at age of two, single testicle, over bites and dying strangely before age two. That’s when the word autoimmune dropped but I did not have the sense to think about it. They were Dutch bred, meaning pure lines of famous top winning English breeders, country of domestic of this breed in Western World. They were never used since at that time my goal was just to have a pair of beautiful Salukis.
As I was occupied in the medical field, I read an article of professor Paablo in the 80ties, who was connected with the University of Munich. He caught my attention as he researched why a certain country in Europe had the highest ranked cancer cases in population. DNA was researched and he found out that the entire population came from a group of less than 10 Eskimo’s and some gypsy’s less than 6. Eskimo’s were related to each other and so were the Gypsies. He concluded that the genetic pole was so small, that actually the entire population was so in and line bred, that had cost change of DNA, a weaker immune system, a small genetic pole and that had lead to the high cancer score of that country.
There was not a lot to study, no open library to learn about the ancestors of our dogs from the beginning. All, I had was the English book of champions and some history books, novels around this breed. I did write thousand and thousands pedigrees by hand to follow lines, if I could find photo’s added them to the pedigrees.
It was easily tracked back that all our stock, even American stock lead back to one ancestor in this breed, the first great British champion Sarona Kelp.
There was no true knowledge at breeders concerning DNA, too shorten gene poles and so on.
All did their best to keep up the old type not even realising that the majority of the stock did not look any more like Sarona Kelp with his longer scalp and hardly pronounced stop and the longer loins.
The breed had changed in very square dogs with plenty of knee bends as if they were going through their knee, short broad heads with lots of stop, barrow chests, and often round bones, round feet, ear leathers. The hip bones were far lower that the dip in the top line, showing the heaviest part of this breed, the carcass. Even if one would show breeders photo’s of the first imports to the Western world and Sarona kelp and point out the difference with their now a day stock, they were kennel blinded and could not see it.
Still, some breeders realized that they needed to expand gene poles, felt the health problems came due too long line and in breeding. Their first out cross giving slightly or major different types were held against them. They went back into line breeding to achieve the common type again and therefore fallen back into the same problems.
It was and still is a problematic topic among breeders! The British kennel club to acknowledge this breed , demanded litters to be uniform, leaving the natural variety in one litter out. All litters from all breeders should look a like.
One can imagine that many whelps in a litter were culled in the beginning to have one single type. It even went that far that colour got involved. For many years the smooth or short hair Saluki was out of favourite and almost lost existence. As they experienced the colour grizle as recessive, grizle salukis were not appreciated for many years. Considered a sign of genetic weak salukis as slightly the term genetic seems to hit breeder’s world.
It also was as some breeders tried to expand gene poles and imported some American stock, they came to a different type, more hind angulations and a different movement, heads without stop but long, totally straight top lines, straighter fronts and so on, higher tail set, a different movement as Afghan’s. A famous breeder just tried to justify that movement in a world famous publishments; saluki movement, saving energy.
So, we see many now a days top winning salukis with a breed untypical movement.
In the 80ties an article was published, concerning movement of this breed by a famous breeder and judge, who had imported an American dog to expand gene poles and improve temperaments; the saluki should move with drive and reach, parallel front movement as parallel hind movement.
( this part did upset many long time breeders and was against all novels ever written about this mysterious breed by the Bedouins and others since their version; As Allah wanted to give them thousands of years a go, a gift to be able to have meat to eat, as hunger did strike them. It was with a full moon, they saw from the sky coming a strange creature, it landed and as fast as the light, passed their tents, disappeared from their view.They all thought they had imagined but they realized they had not. In the sand of the desert in the cold of the night, they saw, three feet prints. The typical, single tracking hind movement of that gift, Allah had sent them. The a gift to catch meat; our gazelle hunter, the Saluki. )
If we would keep to the breed typical hind movement, now a day such a dog would be named, having the fault of narrow hind movement, instead of single tracking hind movement. A fault, considered a weakness.
Lots of all round judges, knowing too many different breed standards, lost the eye, the knowledge of the history of this breed and what’s so breed typical.
I always feel sad, talking with breeders that live for the so-called English type, with the short low hooks, rounder bones, shorter head with too much stop, the square type, not understanding that we in Western world created that type, different, very different from the functional salukis. How can such a low hocks, come above the sand of the desert, how can knees with so much bent, struggling hind legs to carry the carcass of the dog, hip lines lower than shoulder blades or the dip in the top line, how can such a dog rise out of the sand, still have flexibility in the spine to make drive or speed? Where is the, long masculine flexible neck? Have any one ever seen one of these type of salukis with enormous well laid back shoulders and front feet bent inwards as bananas, winning a race track?! No.
Still, I understand the idea of keeping up the old type.
Breeders talking about keeping up the old type, started out in the 40ties, 50ties, 60 ties. When Great Britain was still our example for how this breed should be. Variety of this breed was not allowed, no knowledge of the consequences of long in breeding and line breeding. They only knew this changed type as the Saluki.
But than again, I also feel very sad, so many have forgotten that this should be a modest breed, even when it goes through the ring of honour to win best in show or not. Movement should remain breed typical, modest in everything, single tracking. Never front feet reach out, should pass the point of the nose, head and neck should not be carried straight up as the flashier Afghan but in a less than 45 degree forward, upwards out of the carcass. How sad it is to hear, judges talking that grizle colour is out of fashion. Can it be a breed 10.000 years old, became fashionable? What about these poor salukis where the second tie is much longer than the first tie? We name it politely over angulated, I name it an unbalanced hind movement, which damages the hip joints. A clear sign is the weaker kneecaps, dogs going through their knees and knees are slightly turned out wards. Hip displaces, top lines so straight, one thinks it is a cord for circus clown to perform up. It is sad to hear judges say; the saluki should have a long and elegant neck, bent like a swan. Where remains the strength to take down a gazelle?
Of course we can say, it is a matter of taste but do we realise that these external differences from the thousands of years ago ancestors of our now a day saluki, are also a sign of health problems?
It is hard because we need to blend this exaggerated type to the so- called old fashioned English type to get at least a change to achieve again, the old Sarona kelp type. It seems many think any black and silver dog, is Sarona Kelp. It was not the colour but the construction.
Now, as breeders, we should be a ware that there are genetic inherited problems in this breed.
It is up to each one of us to publish about it, maybe in an effort to help other breeders or not, to hide it for name and fame, consider it normal or acceptable or to find an excuse claiming a genetic inherited disorder was cost due to stress.
1990
Due my first Salukis that were so severe disturbed by temperament and genetics, as El Ubaid’s started the goal was and will remain to breed healthy and good temperament and functional salukis. If winnings were achieved, are being achieved a long with that, it is nice but the pleasure, joy and my conscious remained clean towards this breed.
Genetically or genetically inherited;
It is about time, we stop talking like chickens without heads, know that being born with, in other words, genetically and the very different genetically inherited, meaning came from ancestors and will be passed through to off springs. Genetically inherited is also found by scientist in the genes of a dog, in the DNA of the dog.
B- litter
In my B litter two puppies were put down by vet as they were borne with almost total lack of under jaw.They were drawing trying to drink breast milk.Not because of a less show quality but according the Dutch firm animal protection and kennel club rules, due to the fact they would have died, drowning in the breast milk of their dam. The DKC (Dutch Kennel Club demands scientific medical prove from vets). However my B litter was used over and over and never produced that problem. Therefore the conclusion is to make it was not a genetic inherited problem.
M& G litter
So, I can very easily say what I have bred; I bred in my M and G litter a male that was single testicle. However from both litters, there has been bred and that problem did not occur, even line bred. Therefore we should learn indeed something might appear, when we breed but we have to make sure, it is not genetically inherited. We can’t take the risk to breed with a dog showing a disorder even if we know it was not genetically inherited. I taken my responsibility as the breeder offered the total purchase back in the G litter case and in the M litter partly paid the operation to have the dog castrated to safe its life. In the G litter case the puppy left me with both testicles down in the scrotum and stable but not a single hair on my head would ever consider to blame an innocent purchaser in such a case and start a world wide war against that person or life time hate. My M and G litter have been used, no problems of single testicles occurred or any growth disorder problems even if such a litters were line bred. Meaning it was not a genetically inherited matter.
(Therefore, people that gather together, breeders in denials of disorder stocked they sold to innocent purchasers and get confront ate with the problem and take this bad attitude and contribute a site; claiming in defence of the truth, the el ubaid’s introduction, simply make a fool of their selves. I am too outspoken about these issues concerning my own breeding and concerning dogs I own.)
Growth disorder;
Yes and I had a growth disorder syndrome in two puppies in my G and in my M litter. The total skeleton was de-formed and they were not able to stand or eat and were put down by vets and those reports went as well to the Dutch kennel club. However as I bred out of Guinevere and kennel Khalil and many others out of Genesis, we may conclude, whatever it was, it was not genetically inherited.
Al Caliph
Yes, I did import an Al Caliph male to show and breed from but his second testicle vanished into his stomach due that the cords were too short and he was operated to safe his life. Name of the vet has been published here. I was strongly advised in 1990 by breeders and by vet’s never to use that dog at stud. I taken my financial and emotional lost. I like that chapter to be closed. There is no excuse to blame an innocent purchaser for a misfit we have produced. Denials around this are NOT in defence of the truth
Srinagar Parvati, Cardiologic problems within the breed.
I bought Srinagar Parvati, the deal was I would show her, breed from her, have a litter from her and sent her back to her owner with 1 pick puppy. Also all other puppies sold, half of the purchase price had to go to her breeder. Parvati was the only one in that litter that had lived to that age. All others had died, the breeder had informed me on forehand. Parvati should be bred by El Ubaid’s Bizar on her breeders demand on her next heat. As I returned with Parvati, I ended up in the hospital and not even 3 weeks in the Netherlands Parvati was in heat and mated to Bizar, all in agreement with her breeder. At the echo sound test, 4 weeks after Parvati was mated, we have been fortunate she had remained empty. Parvati was detected with a lethal genetic inherited life threatening heart disorder. Only than her breeder informed me, she had lost quit a number of dogs due to that problem. Her breeder was informed and given all chance to come and pick the bitch up on her responsibility and risk for the bitch her life during the flight. Her breeder gave permission to place the bitch and so I did.
So, there are breeders that test their dogs and breeders that don’t. Any dog detected with this problem should not be bred from. I know the Srinagar lines are behind many dogs, even behind my foundation bitch. It does not mean all Srinagar dogs have this problem or their descendents but an ethical breeder towards the breed does test their dog once a ware that problem is there.
Kennel Lady Hawk with the great Srinagar Glory do test their dogs. A person that owns a Srinagar male and a Srinagar off spring male got very upset around this Parvati issue and went and opened a site El Ubaid’s introduction in defence of the so-called truth denying all these matters, even publishing the university of animal health care in Utrecht and indeed researched and diagnosed a saluki bitch Parvati owned by me but would fall in a defence claiming could have been any other saluki bitch since the specialist had not noted down the chip number.
My goal and my achievements were and are to breed dogs, salukis that are not genetically inherited with any lethal disorder. If because of that all saluki owners and breeders world wide dislike me, has no value to me.
Due to Parvati, I learned again, I had Srinagar Durga immediately fully tested and all her off springs before they left me, got a great cardiologic research and all were found free of this disorder.
That to me is important and I have been blessed with Srinagar Durga.
An other import, genetically inherited dental issues
I came across a problem by a male I imported, who lacked a p1, p2, and p3. Since I have no knowledge of the inherit factor, neither could the university of animal health care help me out on that one. To their opinion, lacking a p1 is often a fact that the p1 is there but since it is almost a useless dental, it is very minor and still hidden in the jaw under the skin, gum. The same could even be the case with the P2 but Not with the P3 and that is a functional dental but of course a dog can live without. However, according to them, I should ask the breeders of this dog’s parents or litter mates had the same problem. If they had only than I should conclude it was genetic inherited! So, I did and they looked in that litter and to their big surprise others litter mates had it as well. I am great full finally some breeders that have been honest.However, maybe due to the almost 36 hours being crated during the flight that male was hyper active and enormous destructive, even in 2010 I am still left with bills of almost 4.000 euro's of matters destroyed by that dog.
So, I turned to the Dutch kennel club, they clearly and Dutch judges state; A complete set of teeth and dentals is demanded. There are also very good and new rules at the DKC, no breeder is allowed to breed with any dog, knowing this dog has disorders. Missing dentals are a disorder, where it comes to the standard of the breed. Still, I called and did write to some breeders around this, I was very surprised Americans did not value it but some major impressive breeders, major winners in Scandinavia, even judges did not value missing dentals. I did not take the risk.
I never bred a saluki missing a dental. I bred one Saluki out my American import Srinagar Durga that seemed to miss a p2. Due to the case of that dog I had imported missing all those dentals, I demanded that saluki bred by me to have an x –ray, to know if that dental was really lacking or hidden under the gum. It was hidden under the gum. I could have it operated but it would leave the dog in pain for weeks and that was not worth for me.
Elektra Helwa, socializing problems but a fertility problem as well.
I imported Elektra Helwa but her temperament was so problematic that it drove not just me and partner crazy but all other dogs as well. Her ovaries were not functioning the way they should, meaning even with medication she would not ovulate, get in season but no ovulation. She was homed with agreement of her breeder and owner to her sire.
Al Gazara’s Byzanthium, geneticly enherited heart problem
I imported Al Gazara's Byzanthium, sire to my Q and I litter. He died 2 years ago, 2008, age 6, of a genetic inherited heart disorder. I published it and informed all owners to dogs out of my Q and I litter to have their dogs cardiologic tested as I did with Srinagar Durga. I got no response beside from kennel Khalil, they tested their dog from my i litter and he was found free of that disorder. Let it be known, Byzantium was called Ghandi, he was so enormous easy and gentle, peaceful that the name Ghandi seemed more fitted to him. I have to say exact the same around my Al Caliph import.
But lets remain honest, I imported Srinagar Durga, Sulaymania Nadjah Ishtar, Kashmani’s Quadham, Qamar von der Irminsul, Elektra Helwa even Aziz Balthasar and litter mate Aziz Batlhsarina. These dogs have contributed my life, friends and family’s life or breeding in a most pleasant way.My Mother now in her 80ties still talks about Tigri (tiger) Aziz Balthasar because she loved his musculinity and problematic Alpha behavior, macho behavior enormously. Still, laughing how every time he would run off with her spectacless and chew them up.We have a Dutch- South American expression; He was a dog to kill, to store behind the wall paper.Meaning; to love and hate.M
Concerning my Aziz imports. Temperament;
I don’t think they should be part of this page but I will honour their breeders statement. However, since my 2 first salukis in the 80ties were two litter brothers, I think that many years later as I imported Aziz Balthsarina and Aziz Balthasar and their breeder blamed me for their Alpha aggressive temperament. Not being experienced enough according to her to handle, control two puppies of the same age. I have to say this, Out of the litter between Aziz Balthasar and Aziz Balthsarina, 10 puppies , all were extremely alpha aggressive. In my A litter, Multi Ch. El Ubaid’s abdull Aziz was problematic Alpha aggressive and so was El Ubaid’s Ashley. I never bred from those two !!!
Where it comes to temperament, to me the Saluki that use to travel free of leash from one regent to the other in country of origins can never be shy, reserved or hysteric but is obedience and affectionate bounded to it’s owner. Yes, alert since they are hunting dogs but aloof they should not be. However the first 16 weeks, are the weeks to be intensively busy socializing your dog, put a stable temperament in there. Hard work, if people don’t have the time to socialize your dogs, don't breed !!! One can only breed from a socialized sire and dam. Especially the dam will be the example of most influence to the puppies in their first 8 weeks.
If we know we are dealing with a mass breeder, a puppy mill, a saluki collector, get your puppy between 8 and 10 weeks to put the correct fundament in there. Realise there are lines that are genetically disturbed where it comes to temperament. I refuse to put up with destructive, disobedience, alpha aggressive dogs, shy, hysteric dogs. I refuse to put up with any dog that is genetically inherited with any disorder.Life time on medication or tranquilizers. I for sure will re-place such a dog, learned my lessons along the way and will take my financial lost.
I have always kept up to maximum 4 salukis within Amsterdam, on the country side max.6.
I am not a dog collector; there are many great breeders and interesting lines. Even if I have to admit the longer one is in the breed the less they get because of knowing the genetics behind the dogs. I prefer people who have the time, that life in the crowded city, no land.
People that share their bed, sofa and daily life with their dog. People that will go shopping, by bus and train with their dog. Take it to friends, to dinners.
Travel to beaches and forest to make dogs run. Have to leave their dogs with reliable friends since they are too busy. The best socializing for a dog instead of meeting my dogs back that lived on an isolated island, not a costume to nothing, shy and aggressive. I can put the fundament in there but the owner has to precede my work.
Autoimmune problems in the breed
The biggest problem in this breed is auto immune problem. However, now a days the cardiologic problem is hitting the breed as hard. I know quite sure I used dogs at stud who’s relatives litter mates or ancestors did have that auto immune problem. One always finds out later because stud dog holders never inform one on for hand.
There is no testing for this problem. One can check certain blood values, liver values and lack of certain proteins and vitamins which can be an indication. If I remember well vitamin b12, a lack of that can be an indication.
When breeders start; I have lines where it never occurred, we can say that does not exist, each line had at least a case or some cases. However a breeder can say and be absolutely correct claiming, the breeder never produced a case. I am one of those blessed, lucky but maybe sensible breeders.
1997 to 2003I lived in fear from 1997 to 2003. Fear that by information I had gotten, my fundament sire, sire to my A litter would have been a case dying at age 7 almost 8!!! This is where the first knowledge came, many universities informed a fact of auto immune disorder; it will at all time appear before age 5, even 2 or 3 if it is a case !!!
So, in 1997 my worries with that information was taken a way from my A, litter but than as genetic science teach us, to have a dominant case, it means BOTH parents have to be recessive carriers.
Still, it leaves one with a major problem if indeed, the sire of my A litter had died age 7 of this disorder, impossible as scientist claim and not a single lab in Finland had any records of this and never the evidence was sent to me. Still, it left me in doubt.
Let's just imagine that fairy tale would be true, it would mean some in my A litter would be carriers and some would be free out from this genetic disorder. There is no testing, so we can make sure if we are a ware and know lines and genetic, too out cross and keep our lines healthy, breed a way from a lethal problem. For once I hope people understand I am talking about my Breeding!!!
The sire to my A litter Lux and Sf Ch. Aziz Balthasar, ( Sw.Sf.Race Ch.Kirman Osman x Maleke)
I heard this news around him end 1997. I had bred my B litter in 1995 and my C litter in 1996.My C litter were seven puppies and since I was not a ware, I had doubled on the lines of Maleke, the dog I used at stud was going back to Maleke's litter mate Mohkam. As I contacted universities and they contacted the ivis, I was pointed out again, even if maleke and mohkam were litter mates maleke might have been a carrier but that does not automatically made Mohkam carrier. So, I researched at breeders and found out there had never been case after Mohkam. But what if breeders were hiding???
So, my sleepless night went on, but in 2010, my entire c litter reached age 10 to 13 almost 14, all in great health.
Had I been so lucky???
As I bred my D litter, it seemed now I should absolutely have a case this was in 1997 before the news came to me. There it seemed I used a dog at stud who’s litter mates would have it and I not knowing doubled and doubled on lines I simply admired, quality of dogs but as many said, than I was collecting problems and my D litter would explode in my face. Well in 2010 my D litter 6 (1-5) all reached age 10 and some are still going strong age 13 as my own Multi ch. El Ubaid's Dignity, now a days the grand dam in the house.
Lucky again ??
By 1999, I wanted to know if this is pure luck, due to circumstances I ended up and used an offspring to Sf.Sw.Race Ch.Kirman Osman to Kirman Osman's granddaughter, my own multi ch. El Ubaid's Anastasia. 9 puppies. Age 10 all are a life and healthy.
Luck again? Can be!! Because even if a bitch that is known to be recessive carrier, not each ovum will carry the gene for this genetic inherited disorder and not every semen cell of a dog known to carry this disorder, will have the gene of this disorder.
That's why even in very uniform litters we still might have different colours ;-))
For this reason some breeders very a ware their dog is a carrier, will continue to pay Russian roulette and take the risk, leaving behind them a path of suffering dogs, emotionally hurt innocent purchasers, who if they dare to speak out will be attacked by such a breeders or worse.
But being tired of all the medical researches I had done, thyroid, willebrandt disorder and so many more, I changed lines, produced my F litter, in 2000 now age 10 not a single case. Yes, a dog to exported to Austria or Switzerland died age 2 and of course dog scene start sending me e-mails, the dog would have died of auto immune disorder. I still thank his owner Mrs. Veltner for sending me the medical rapport, her dog was simply poisoned.
So, the sire of my F litter was from Belgium, actually taking back the lines of my fundament bitch, Sulaymania Nadjah Ishtar.
My G litter now age 8 whelped in 2002 now age 8, are all healthy.
H litter total; out cross, all healthy age 8
I litter again total out cross out of my beloved Dignity, they are age 8 as well and all healthy, at least no autoimmune disorders.
My M litter, I used Multi Ch. El Ubaid's Dignity to dog, of which many claimed he should be a carrier but they are age 5 now and not a case!!!
I doubled my lines, my D litter to my E litter, tripled on Kirman Osman, doubled on Maleke, doubled on all accused lines,doubled on Aziz Balthasar,not a single case.
Now, in 2010, I have to sit back and relax, the dogs I used in My breeding, dogs I used at stud from others even how accused they have been and maybe even proven produced problems at other kennels, they never did in my breeding.
I used from my own breeding Anastasia and doubled on her frequently and not a single problem used her to accused lines, not a single problem. This is where one can say, NO this is NOT luck the bitch was simply not a carrier.
I used Aphrodite to a dog very accused to have problem but the first one in that litter died age 7, one and 10, one at 11 and two went on age almost 15. This was my B litter.....all achieved ages in full health meaning passing that 5 years age with in a dominant auto immune case will show it selves.
I used Multi ch. El Ubaid's Cadiya not a single case, I used El Ubaid's Cheddah and Bizar, not a single case and I even used Al Gazara's Byzanthium, Srinagar Durga and a Él Ubaid's Djoser's son not a single case. They have not been carriers.
I can only conclude the dogs I used, out of my own breeding and not living with me and dogs from other breeders at me never produced a single case.
The El Ubaid's Bitches used by me; Anastasia, Aprodite, Cadiya, Empress, Guinevere, never produced a single a case, meaning the way I have bred and doubled on certain lines for sure they have not been carriers.
When gossips or accuses like this hit us, it takes us many years to find out the truth, to get the knowledge and experiences. It cost us sleepless nights, research and medical research, lots of high phone bills and so many hours e-mailing.
I was told but evidence was never given even when asked for, that multi ch. El Ubaid's Abdull Aziz would have produced cases at kennel Aziz, as he did stude, the sister to his father. This will always will make me doubt that statement since his sire and the same litter sister, produced a litter of 10 puppies and all reached age 10 in full healths, no auto immune disorders. I have to deal with facts, proven facts and not gossips. Still, I like to give the benefit of the doubt and all I can than say; Well, than if this is true, I am happy that even with his great quality, I never used him at stud.
I know a breeder that bred one single litter out of a bitch from my D litter, against my advice to a dog which was known to be carrier. I pointed out her bitch could be a carrier but she went a head and not even 2 years later the medical evidence, diagnose was sent to me from one of the puppy owners out of that litter, that her bitch had suffered enormously since age 9 months and died due to an autoimmune disorder. I still compliment that breeder, she realized the problem came from both sides and never used her marvellous bitch again. Of course she could have outcrosses to different lines but with the general attitude of hiding facts, how will you ever know if the dog you use at stud is not a carrier as well?
I think and feel dogs or bitches that have proven produced cases should not be used any more in any breeding program simply to protect the breed, we claim to love. That's ethical. Let's say it one more time, scientifically proven; it takes a recessive carrier dam and recessive sire to have a dominant case. People claiming they got a case due to a stud dog that was bred by somebody else, are hiding their head in the sand.
I also like to repeat this, in such litter, it does not mean all will be dominant cases or carriers, some will not be. How will we know? I would say by the way I bred, doubling on lines to find out and know for sure.
We can use many excuses why an autoimmune case appeared, stress, divorces, environment, and food and so on. The fact remains the dog had a genetic inherited weak immune system and of course such a dogs can easily earlier-sooner get sick and diagnosed with the autoimmune disorder due to stress or whatever.But let it be known the dog would be diagnosed with that disorder any way before age 5 !!
It does not mean, the crate or the stress created the problem, the problem was already there and sooner or later the dog would have died due to this disorder. The circumstances only did speed up the time of collapsing of the immune system
I went into out crossing due to my great import of Srinagar Durga lines known free of those disorders and I am very satisfied with the healthy litters she produced. I simply did outcrosses to expand the gene poles of my healthy stock. No more !!
Concerning my B litter.Now there is one rumour I would like to stop since I am a too out spoken breeder and before talking about the problems of someone else I prefer to publish around the problems in my own breeding.
In my B litter age 10 months, all dogs that taken part at a certain show in Germany, got infected of the virus hepatitis canine infectious, it was not an indication but a proven scientific diagnose. Bellophor, Barnabas, Bahdiva and Blakka Perka. Due to that all these dogs lost their complete coat a month after gotten infected. They all died years later of an organic defect due to this VIRUS.
However the two that did not attended that show, Bizar and Batiste, never got infected and lived up to 13 and 14 years old. They also never produced a problem!! Yes, a German breeder attended that show as well. She lost 5 of her salukis due to this Virus.
Let this be a warning for those that are against vaccinations; It is needed, even with an vaccination, your dog is never 100% guaranteed protected against this dramatic virus.
Understand well,; A VIRUS, is NOT something genetic, it is not in the DNA .However any dog or bitch that gets this virus will die younger, due to the fact that this virus destroys- damages, muscles, organs. There came in 2003, due to the dead of Ch.Jw.Winner1996 Top Saluki number one 1996 El Ubaid’s Bellophor a lot clear to me.
Any dog infected by this virus will while on medication or even on medication carry this virus in his liquids, only for sure after 18 months, we can be sure the virus in the dog has disappeared , is killed, until than, this dog can remain infecting others, his own kids or others. Such a dog should NOT be used at stud during those 18 months. His semen, liquid, so not within the semen cells but the liquid that comes a long with that semen as in it is saliva the virus is still there. Which means puppies of a dog within this period will be born infected, we will not notice but they will die at young age before 3 or 4 of organic damage. So, it might look like a genetic inherited disorder but it is a virus.
When a dog not born without this virus, get infected, it’s eyes get yellow the gums lose colour but the dog loses the entire coat. I met a dog like that on the European winner show in Denmark, 1997, it was a deep black grizle, had changed in almost a crème with simply a hardly to notice grizle marking left. His owners did not want to speak but claimed a new shampoo and there had been an infection.
Of course the question raises, why some get born infected and some not. We should consider not all ovum’s are at the same point- level, the semen cells, lose the liquid they are in as their energy, (their tail is their energy pushing them forward) while travelling towards the ovum. Those ova at lower level will get the semen cells that had a short travel to do with lots of the liquid carrying the virus. Those very up high, will get semen cells without liquid. The unversity showed me how it works; take some young frogs still with tale as fish put them in a bowl with egg jump( the egg jump sould be considered the liquid with the virus, the young frogs considered the semen cells) let them swim in the egg jump, it will stick to their body than take the frogg fish and throw them in a bowl (aqauria) with clean water, the longer they swim the less of the egg jump stay on their body and after a long time of swimming nog egg jump (virus) will be on their body any more.
I only went in this matter as a German breeder in 2010, not knowing the issue but would confirm me as if she was vet, the Ch.Winner, El Ubaid’s Barnabas and a certain kennel in Germany produces auto immune problems. This case was researched by the University of Animal health care, DNA specialist and specialist on auto immune disorder Prof. Slappendel.
I also went with the two laboratory test of Barnabas his children to the university. None of this lab test claimed an auto immune disorder. The blood values only showed poisoning, which could come by a disorder of kidney or liver but it could be simply poisoning. Besides those Barnabas off springs died age 7 meaning past the scientific age of 5 which is the maximum age for this disorder to come up as a dominant case. Thosae dog were not indicated or diagnosed as cases according to their lab test.
Due to Bellophor’s dead and a scientific pathologic rapport, we took the possibility of the effect of the virus hepatitis canine infectious. Barnabas after that 18 month period produced a litter in a different kennel in Germany and not a single problem. Bellophor produced years later a litter in Belgium, not a single problem.
In 2007, I asked kennel D’alfabia personally if they had any problems in that litter age 8 than if not older and not had a single problem. I realise that litter was never used because of certain politics, demands made on them, if they continued with that litter a certain stud dog they wanted to use from a good friend-family member of theirs would not be available at stud.
Never the less, I never used Bellophor or Barnabas at stud but Bizar and he never produced a problem. Luck? Well, I don't consider myself such a lucky person.
There exist no lines world wide that never had genetic lethal disorders. I did not breed it myself but for sure in the ancestors of dogs I used in my breeding program it happened.
Breeding healthy salukis is keeping these matters recessive but not denying their existence.
I will not claim always something can happen as an excuse. I don’t like to take the risk.
However, as long as we use lines, we have no experiences with, we are on risk, since we can’t count on the honesty of the majority of breeders and they might be focused on something else much more than breeding “healthy “salukis.
By the way I used; Sulaymania Nadjah Ishtar, Anastasia, Dignity, Empress, Durga, I can guarantee they have been free of any genetic disorder!!! If I used an El Ubaid’s bred male he was on such a high age and guaranteed healthy.
I never used a male at stud owned by me or living at me. I always photographed each mating. I therefore can guarantee as well, the pedigrees in my breeding are 100 % correct.
I have tested my dogs, owned by me on the von Willebrandt disease, thyroid, liver, kidney functioning, and regular protein and vitamins check up. There are simply breeders with different goals and ideas.
One request, I have learned not to sent my rough written articles to so-called friend breeders.
As I did write my first health issue, a negative breeder, new eager to win made from a 40 page essay written by me an over 140 page essay and added a lot to it, to steer breeders up world wide.
I never got the chance to analyse all my notes and try to make it compact.I think I finally succeeded 12 pages for those interested to read it.
Once again, let it be clear, this is around my breeding and dogs owned or once owned by me.
It is an afford to explain my breeding, my thoughts and experiences, my researches. To some it will remain a too up front part, too outspoken, to some too negative and bitter but to some it might be very interesting and maybe even a help.
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